As discussed following are 2 documents I mentioned for your consideration
and application. So far 5 people, that I know, have tired it and
have had very good results.
1) Linus Pauling Therapy for Heart Disease is at http://www.paulingtherapy.com/
and copiously describes a non toxic, cost effective Orthomolecular
approach using vitamin C and amino acids Lysine and Proline.
16] According to the Pauling/Rath 1994 United States patent, the
amino acid lysine (lysine analogs), along with vitamin C and other
antioxidants (e.g. Co-Q10, vitamin E and vitamin A), can, in sufficient
concentration, inhibit Lp(a) binding to exposed lysine residues.
Proline residues are also exposed by lesions in blood vessels. Later
experiments showed that proline as well as lysine, with vitamin
C, other amino acids and antioxidants, in oral amounts well past
what is needed for prevention, becomes a solvent by inhibiting the
binding of Lp(a). A binding inhibitor augmented with vitamin C can
stop and apparently even reverses some plaque formations. Pauling
and Rath even have a second U. S. patent for using these binding
inhibitors as solvents to melt atherosclerotic plaques from human
organs during organ transplants. The organ is dipped in the Lp(a)
Binding Inhibitor solution and the plaques melt away.
U. S. Patent # 5,278,189 is for the prevention and treatment of
occlusive cardiovascular disease with vitamin C and substances that
inhibit the binding of lipoprotein-(a). The full patent is available:
If you prefer I can email it as a pdf file attachment to you.
Backing this up with Vitamin B6 is described in the attached Joe
paper "Vitamin B6: The overlooked Key to preventing heart attacks"
ABSTRACT: Vitamin B6 (pyridoxine) opens the door to eliminating
the 20th century's epidemic of heart attacks, cardiac arrests and
strokes. Although shunned by the researchers who receive the bulk
of heart disease research funding, it is creating excitement among
a growing number of investigators.
In this article relevant bits of B6's history are presented to show
how it can prevent heart attacks with almost no side effects from
moderate amounts. This article will also integrate the effects of
vitamin B6 deficiency with Mathias Rath and Linus Pauling's theory
(blaming heart attacks on deficient vitamin C and excess Lp(a) and
Bruce Lipton's histamine theory into a general theory of atherogenesis.
As usual any effective products such as amino acids which are very
effective for many diseases and essentially non toxic (far less
toxic than even salt) are banned illegally by Health Canada to protect
their pharmaceutical friends! So you will have get them from the
states - 250 (500 mg) tablets of lysine are available for approx
$5 from US Walmart stores. The underground cost in Canada is over
$50 courtesy of Health Canada.
Reduction of Lipoprotein(a) in Postmenopausal Women
As a general surgeon who is interested in the prevention of postmenopausal
heart disease and in particular in the reduction of lipoprotein(a)
[Lp(a)], I read with great interest the recent review by Dr Mosca1
regarding the role of HRT. Rapid progression of arteriographically
determined coronary artery disease has been significantly more common
in subjects with Lp(a) levels higher than 25 mg/dL.2 Approximately
33% of the population have elevated levels of Lp(a) (>25 mg/dL),
a condition that is an independent risk factor for coronary artery
disease.3, 4 The treatment currently recommended for postmenopausal
women with Lp(a) levels higher than 25 mg/dL consists of a combination
of HRT and niacin.4 The adverse effects of niacin use are flushing,
headache, and liver dysfunction. I am a 53-year-old woman with a
significantly elevated level of Lp(a) (27 mg/dL), but I found that
I had to stop taking niacin primarily because of headaches. Thus,
after a thorough review of the literature, I began to follow the
advice of Linus Pauling. For individuals who have an Lp(a) level
higher than 25 mg/dL and a family history of heart disease, the
recommendation is to take 3 g/d of both ascorbic acid and L-lysine
monohydrochloride.5 After 6 months of this regimen, with no adverse
effects, my Lp(a) level decreased to 14 mg/dL, a reduction of 48%.
The Lp(a) testing was done by the highly reliable Lawrence Berkeley
National Laboratory/Berkeley HeartLab Department Technology Transfer
program. The theory is that lysine is an Lp(a)-binding inhibitor
and thus blocks the Lp(a) attachment to the arterial blood vessel
wall and that ascorbic acid helps to repair the collagen injury
to the blood vessel and acts as an antioxidant.5-7 Currently, pilot
studies are being conducted on the Pauling therapy of elevated levels
Kathie M. Dalessandri, MS, MD
Point Reyes Station, Calif
1. Mosca L. The role of hormone replacement therapy in the prevention
postmenopausal heart disease. Arch Intern Med. 2000;60:2263-2272.
2. Terres W, Tatsis E, Pfalzer B, Beil FU, Beisiegel U, Hamm CW.
angiographic progression of coronary artery disease in patients
elevated lipoprotein(a). Circulation. 1995;91:948-950. MEDLINE
3. Bostom AG, Cupples LA, Jenner JL, et al. Elevated plasma lipoprotein(a)
and coronary heart disease in men aged 55 years and younger: a prospective
study. JAMA. 1996;276:544-548. MEDLINE
4. Superko HR. Did grandma give you heart disease? the new battle
coronary artery disease. Am J Cardiol. 1998;82:34Q-46Q. MEDLINE
5. Pauling L, Rath M. Solution to the puzzle of human cardiovascular
disease: its primary cause is ascorbate deficiency leading to the
deposition of lipoprotein(a) and fibrinogen/fibrin in the vascular
wall. J Orthomol Med. 1992;6:125-133.
6. Rath M, Pauling L. Hypothesis: lipoprotein(a) is a surrogate
ascorbate. Proc Natl Acad Sci U S A. 1990;87:6204-6207. MEDLINE
7. Pauling L. The Last Interview [videotape]. Lisle, Ill: Intellisoft
Multimedia Inc; 1994.
(Arch Intern Med. March 12, 2001;161:772-773,)
Reduction of Lipoprotein(a) in Postmenopausal Women